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International Journal of Research in Paediatric Nursing

P-ISSN: 2664-1291, E-ISSN: 2664-1305, Impact Factor: RJIF 5.6
International Journal of Research in Paediatric Nursing
Printed Journal   |   Refereed Journal   |   Peer Reviewed Journal

2021, Vol. 3, Issue 2, Part A

Moyamoya disease: A case report

Dr. Geetha C

Moyamoya. Disease may be a rare, progressive disease caused by blocked arteries within the basal ganglia at the bottom of the brain and form of cerebrovascular disease that begins with an obliterative vasculopathy and progresses to a compensatory proliferative vasculopathy. The term Moyamoya (MM) was coined by Suzuki and Takaku in 1969 to describe the appearance of small collaterals in the thalamus and traversing the basal ganglia due to progressive stenosis. In a young patient, Moyamoya disease is an arterial disorder that causes a stroke. This is a chronic condition characterized by bilateral stenosis and occlusion of the arteries surrounding the Willis circle, as well as prominent arterial collateral circulation. It was discovered in Japan for the first time. Classic angiographic findings of stenosis or occlusion of the circle of Willi's vessels are used to diagnose it. I describe a 9-year-old male child who was diagnosed with weakness at the age of 6-months. He had remitting slurred speech and right-sided facial droop for three weeks prior to this admission, and he had head trauma after falling from a tree. An electroencephalogram and a computed tomography (CT) of the head were used to assess him. After four days in the hospital, the child improved and he was discharged. The child's condition improved gradually as medications, physiotherapy, and speech therapy were continued, and parents were counseled about the child's condition, treatment, and management.
Pages : 16-20 | 706 Views | 327 Downloads


International Journal of Research in Paediatric Nursing
How to cite this article:
Dr. Geetha C. Moyamoya disease: A case report. Int J Res Paediatric Nurs 2021;3(2):16-20. DOI: 10.33545/26641291.2021.v3.i2a.69
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